Dystrophic Intracranial Calcification Accompanying Varix of the Dural Sinus and Arteriovenous Malformation
Makaleler > Tam Makale · 2018
Yazarlar: Mehmet Haydar Atalar, Rıfat Nuri Şener, Bülent Yıldız, Özlem Kayım Yıldız
Turkish Journal Of Neurology, Uluslararası, Scopus, 2018, cilt 24, ss 277-278
DOI: 10.4274/tnd.32396
Özet
Dear Editor, Intracranial calcifications may occur for various reasons and manifest with a variety of symptoms and signs.They are commonly encountered in neuroradiology daily practice.The main causes of calcification include hypoparathyroidism, pseudohypoparathyroidism, Fahr disease, Wilson disease, mitochondrial encephalopathies, neoplasms, anoxia, carbon monoxide and lead poisoning, and radiotherapy.They may also rarely develop secondary to an underlying vascular pathology, as in our patient (1,2,3).A 15-year-old boy was admitted to the emergency service following a generalised tonic-clonic seizure.No specific neurologic deficit was detected in a physical examination.Routine hematology, liver function tests, renal and thyroid function tests, and serum calcium and phosphorus levels were all normal.An urgent computed tomography (CT) scan was performed.A CT of the brain showed extensive gyriform calcifications located at the corticomedullary junction (Figure 1A, 1B).There was a calcified varix (V) connected with the superior sagittal sinus.For a detailed view of the detected findings in CT, magnetic resonance (MR) imaging and MR angiography (MRA) were performed.MR examinations showed dilated middle cerebral, and posterior cerebral arteries also supplying the arteriovenous malformation.T2-weighted and post-contrast T1weighted MR images showed the V, as well as MRA (Figure 1C).Conventional angiography demonstrated a nidus of a parenchymal arteriovenous malformation.In addition, the cerebellum was Ad dress for Cor res pon den ce/Ya z›fl ma