Thrombin activatable fibrinolysis inhibitor in Behcet's disease

Makaleler > Tam Makale · 2005

Yazarlar: A Donmez, Kenan AKSU, Handan AK, Gökhan KESER, S Cagirgan, SB Omay, V Inal, Hikmet Hakan AYDIN, M Tombuloglu, E Doganavsargil

THROMBOSIS RESEARCH, SCI Expanded İndekslerine Giren Dergi, Uluslararası, 2005, cilt 115, ss 287-292

DOI: 10.1016/j.thromres.2004.09.010

Özet

Introduction: Thrombin activatable fibrinolysis inhibitor (TAFI) is a procarboxypeptidase downregulating plasmin formation, thereby causing a tendency for thrombosis development. Since, Behcet's disease (BD) is a systemic vasculitis, which is commonly complicated by arterial and venous thrombosis, we aimed to find out plasma TAR levels in BD, compared with healthy controls. We also searched whether plasma TAFI levels were significantly different between Behcet's subgroups with and without thrombosis.

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